## Description DDFT tendinopathy refers to degenerative or traumatic injury to the deep digital flexor tendon, most commonly occurring in the region where the tendon glides over the navicular bone within the podotrochlear apparatus. Lesions can include core lesions, dorsal surface fibrillation or tears, and adhesions to the navicular bursa or bone. This is one of the most significant soft tissue contributors to caudal heel pain in horses. The condition often develops from repetitive overload, particularly in horses with conformational predispositions or those engaged in high-impact disciplines. It frequently coexists with navicular bone changes and bursal pathology, making it part of the broader podotrochlosis complex. ## Causes / Risk Factors Repetitive high-impact loading; conformational faults that increase DDFT tension (long toes, low heels); poor hoof balance; previous navicular pathology; inadequate conditioning; genetic predisposition in some breeds and lines. ## Diagnosis Clinical examination and diagnostic analgesia; ultrasound of the pastern and fetlock regions; MRI is the gold standard for identifying core lesions, dorsal tears, and adhesions within the foot; radiographs to assess concurrent navicular bone changes. ## Treatment Initial rest and controlled rehabilitation; corrective farriery to reduce DDFT tension (rolled toes, heel support); systemic anti-inflammatories; intralesional or bursal injections of biologics (PRP, stem cells); shockwave therapy; navicular bursoscopy for debridement of dorsal tears and adhesions in selected cases; palmar digital neurectomy as a last resort in chronic cases with confirmed limited tendon damage. ## Prognosis Variable and dependent on lesion severity and location. Mild core lesions or dorsal fibrillation often respond well to conservative management and farriery. Extensive tears or chronic adhesions carry a more guarded prognosis for return to high-level performance. Many horses can be maintained comfortably for lower-level work with ongoing management. ## Prevention Proper hoof balance and regular farriery; gradual conditioning with attention to surface; maintaining appropriate body condition; early intervention when subtle gait changes are noticed. This information is for educational purposes only. Always consult with a licensed veterinarian for diagnosis and treatment of your horse.
Chronic or intermittent forelimb lameness; positive response to palmar digital nerve block; shortened stride with toe-first landing; pointing at rest; worsening on hard surfaces and circles; pain on deep palpation of the heel region in some cases; effusion of the digital flexor tendon sheath in pastern-level injuries
## Description DDFT tendinopathy refers to degenerative or traumatic injury to the deep digital flexor tendon, most commonly occurring in the region where the tendon glides over the navicular bone within the podotrochlear apparatus. Lesions can include core lesions, dorsal surface fibrillation or tears, and adhesions to the navicular bursa or bone. This is one of the most significant soft tissue contributors to caudal heel pain in horses. The condition often develops from repetitive overload, particularly in horses with conformational predispositions or those engaged in high-impact disciplines. It frequently coexists with navicular bone changes and bursal pathology, making it part of the broader podotrochlosis complex. ## Causes / Risk Factors Repetitive high-impact loading; conformational faults that increase DDFT tension (long toes, low heels); poor hoof balance; previous navicular pathology; inadequate conditioning; genetic predisposition in some breeds and lines. ## Diagnosis Clinical examination and diagnostic analgesia; ultrasound of the pastern and fetlock regions; MRI is the gold standard for identifying core lesions, dorsal tears, and adhesions within the foot; radiographs to assess concurrent navicular bone changes. ## Treatment Initial rest and controlled rehabilitation; corrective farriery to reduce DDFT tension (rolled toes, heel support); systemic anti-inflammatories; intralesional or bursal injections of biologics (PRP, stem cells); shockwave therapy; navicular bursoscopy for debridement of dorsal tears and adhesions in selected cases; palmar digital neurectomy as a last resort in chronic cases with confirmed limited tendon damage. ## Prognosis Variable and dependent on lesion severity and location. Mild core lesions or dorsal fibrillation often respond well to conservative management and farriery. Extensive tears or chronic adhesions carry a more guarded prognosis for return to high-level performance. Many horses can be maintained comfortably for lower-level work with ongoing management. ## Prevention Proper hoof balance and regular farriery; gradual conditioning with attention to surface; maintaining appropriate body condition; early intervention when subtle gait changes are noticed. This information is for educational purposes only. Always consult with a licensed veterinarian for diagnosis and treatment of your horse.
Chronic or intermittent forelimb lameness; positive response to palmar digital nerve block; shortened stride with toe-first landing; pointing at rest; worsening on hard surfaces and circles; pain on deep palpation of the heel region in some cases; effusion of the digital flexor tendon sheath in pastern-level injuries