## Overview Tibialis cranialis myopathy is acute damage to the muscle on the front of the gaskin that flexes the hock. It can result from extreme exertion or compartment syndrome after prolonged recumbency. Signs include swelling and pain over the craniolateral gaskin, inability to flex the hock, heat, and dark urine if severe myoglobinuria occurs. Urgent veterinary attention is required. ## Causes Overexertion or post-anesthetic compartment syndrome. ## Diagnosis Clinical signs, elevated muscle enzymes, and ultrasound. Dark urine indicates myoglobin release. ## Treatment Aggressive fluid therapy, pain control, and fasciotomy if compartment syndrome is present. Prognosis depends on severity. ## Recovery and Prognosis Variable. Mild cases recover. Severe muscle necrosis has a poorer outlook. ## Prevention Proper padding during anesthesia and gradual conditioning.
swelling and pain over the craniolateral gaskin; inability to flex the hock; heat; dark urine if severe
## Overview Tibialis cranialis myopathy is acute damage to the muscle on the front of the gaskin that flexes the hock. It can result from extreme exertion or compartment syndrome after prolonged recumbency. Signs include swelling and pain over the craniolateral gaskin, inability to flex the hock, heat, and dark urine if severe myoglobinuria occurs. Urgent veterinary attention is required. ## Causes Overexertion or post-anesthetic compartment syndrome. ## Diagnosis Clinical signs, elevated muscle enzymes, and ultrasound. Dark urine indicates myoglobin release. ## Treatment Aggressive fluid therapy, pain control, and fasciotomy if compartment syndrome is present. Prognosis depends on severity. ## Recovery and Prognosis Variable. Mild cases recover. Severe muscle necrosis has a poorer outlook. ## Prevention Proper padding during anesthesia and gradual conditioning.
swelling and pain over the craniolateral gaskin; inability to flex the hock; heat; dark urine if severe