## Overview Bone spavin is osteoarthritis (degenerative joint disease) of the distal tarsal joints, principally the distal intertarsal and tarsometatarsal joints. Repeated compression and rotation damage cartilage, narrow the joint spaces, and stimulate new bone formation. The result is progressive hind-limb lameness that is one of the most common causes of performance limitation in sport horses. ## Causes Repetitive concussion, conformational faults (straight or sickle hocks), and athletic activities that heavily load the hocks (jumping, dressage, western performance) are the main drivers. Mineral imbalances have been suggested but evidence is limited. ## Diagnosis History of gradual hind-limb lameness that may warm out of, positive flexion tests, and a characteristic “stabbing” or short stride are suggestive. Radiographs show joint-space narrowing, sclerosis, and periarticular new bone. Diagnostic analgesia of the distal tarsal joints confirms the source of pain. ## Treatment Non-steroidal anti-inflammatory drugs, intra-articular corticosteroids, corrective shoeing, and workload modification are first-line measures. Chemical or surgical arthrodesis (facilitated ankylosis) can be considered when medical management fails and the goal is pain-free fusion of the low-motion joints. ## Recovery and Prognosis Many horses remain useful with medical management. Complete fusion, whether spontaneous or surgical, can restore comfort. Advanced bilateral disease may ultimately limit athletic career length. ## Prevention Selection for good hock conformation, balanced shoeing, and sensible training progression reduce risk but cannot eliminate it in high-level athletes.
gradual hindlimb lameness; shortened stride or toe dragging; positive hock flexion test; bony enlargement on the medial lower hock in advanced cases; stiffness after rest
## Overview Bone spavin is osteoarthritis (degenerative joint disease) of the distal tarsal joints, principally the distal intertarsal and tarsometatarsal joints. Repeated compression and rotation damage cartilage, narrow the joint spaces, and stimulate new bone formation. The result is progressive hind-limb lameness that is one of the most common causes of performance limitation in sport horses. ## Causes Repetitive concussion, conformational faults (straight or sickle hocks), and athletic activities that heavily load the hocks (jumping, dressage, western performance) are the main drivers. Mineral imbalances have been suggested but evidence is limited. ## Diagnosis History of gradual hind-limb lameness that may warm out of, positive flexion tests, and a characteristic “stabbing” or short stride are suggestive. Radiographs show joint-space narrowing, sclerosis, and periarticular new bone. Diagnostic analgesia of the distal tarsal joints confirms the source of pain. ## Treatment Non-steroidal anti-inflammatory drugs, intra-articular corticosteroids, corrective shoeing, and workload modification are first-line measures. Chemical or surgical arthrodesis (facilitated ankylosis) can be considered when medical management fails and the goal is pain-free fusion of the low-motion joints. ## Recovery and Prognosis Many horses remain useful with medical management. Complete fusion, whether spontaneous or surgical, can restore comfort. Advanced bilateral disease may ultimately limit athletic career length. ## Prevention Selection for good hock conformation, balanced shoeing, and sensible training progression reduce risk but cannot eliminate it in high-level athletes.
gradual hindlimb lameness; shortened stride or toe dragging; positive hock flexion test; bony enlargement on the medial lower hock in advanced cases; stiffness after rest